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Numerous studies have demonstrated that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to involve down-regulation of cancer stem cells. This may account for the long-term effects observed in certain patients suffering from advanced HCC. However, the reason behind AM EMF's effects on cancer patients is not clear.
Effects on the effects of AM RF EMF on HCC tumor growth in vivo were examined in mice. The tumours were classified in three different groups. First, the group that was unaffected to RF EMF. Second group members were exposed RF EMF at frequencies similar to that of humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on the tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, the tumours treated with RCF did not show any evidence of tumour shrinkage.
The mechanism of cancer-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is caused by CACNA1H, a protein which regulates the Ca2+ influx specific to tumors. Great post to read suggest that CACNA1H could have more broader implications for treatment and diagnosis of various cancers.
The tumours in those in the group that were unaffected EMF from radiofrequency, and fed a standard mouse diet. The tumors in the HCCMF group were treated with Huh7 cells after they were between five and seven weeks old. The tumors were removed after they had a high burden.
The tumors of the three groups also displayed distinct growth curves. The HCCMF-treated tumors saw a significant decrease in tumour size after 8 weeks. However, the tumors treated with RCF didn't show signs of shrinkage. The difference was significant. The tumours treated with RCF were able to show necrosis, which is typical in tumours that are exposed to RCF. The possibility is that this necrosis is caused by a lack of oxygen in the more invasive tumors.
In https://note1s.com/notes/0O7KAR6 , the results indicate an AM-RF EMF has anticancer effects in vitro and in the vivo. A number of studies have proven that AM RF EMF produces measurable reduction in tumours within HCC patients. There is a possibility that AM RF EMF causes these effects due to CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the development of HCC tumours in vivo.