Whether you live in a house or apartment or just would like to ensure your home is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to reduce your electronic device use. You can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another way to shield your house from EMF radiation would be to put up an RF shielding canopy. It is a type of net that contains EMF shielding. It's used to stop EMFs from entering a space. Another option is to get your home fitted with a conductive enclosure. These devices are called Faraday cages.
A number of studies have proven studies have shown that nonionizing EMF produces antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This could be the reason for the long-term responses seen in some patients with advanced HCC. However, the mechanism behind AM RF EMF's effect in cancer patients is not yet clear.
emf blocker from AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumors were split into 3 groups. First, the group that was unaffected RF EMF. The second group was exposed RF EMF at a frequency that is similar to that used in humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on tumours was compared to that of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of shrinkage of the tumor.
The mechanism behind cancer-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H may have broader implications for treatment and diagnosis of different cancers.
The tumors in the control group were not exposed EMF from RF, and fed a normal mouse diet. The tumors in the HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours from the three groups also displayed distinct growth curves. The tumors treated with HCCMF saw a significant reduction in size of the tumor after eight weeks. However, emf blockers treated with RCF showed no reduction in size. The difference was highly significant. The tumours treated with RCF had necrosis, which is common in tumors that have been exposed to RCF. The possibility is that the necrosis is caused by an absence of oxygen in the larger tumours.
In summary, the results suggest an AM-RF EMF exhibits anticancer properties in vitro as well as in live. Check over here of studies have proven the fact that AM RF EMF produces measurable reduction in tumours within HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the development of HCC tumours in the vivo.