No matter if you live in a house or apartment or just need to ensure that your house is free from EMFs, there are a number of methods to reduce exposure. One of the easiest is to restrict your electronic device use. Learn more is also possible to use EMF block paint to stop EMF radiation from entering your house. Another way to shield your home against EMF radiations is to put up a shielding canopy for RF. This is a fabric of net that contains EMF shielding. It is utilized to block EMFs from entering rooms. Another alternative is to have your home equipped with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown that the non-ionizing RF EMF produces antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation the cancer stem cell. This could be the reason for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism of AM RF EMF's effect in cancer patients is not clear.
Aspects from AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. First, the group that was unaffected RF EMF. The second group was exposed to RF EMF at frequencies similar to the frequency used by humans. The third group was exposed the RF EMF in HCC-specific frequencies. The effects of HCCMF on tumors was compared to that of RCF. The results revealed that tumors treated by HCCMF had significant shrinkage. However, block emf treated with RCF showed no evidence of tumour shrinkage.
The mechanism of tumor-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated through CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have more broader implications for the treatment and diagnosis of many cancers.
The tumours in the controls were never exposed RF EMF, and were fed a standard mouse diet. The tumors in those in the HCCMF group were injected with Huh7 cells when they were between five and seven weeks old. The tumours were then euthanized after they had a high burden.
The tumours from the three groups also displayed different growth curves. The HCCMF-treated tumors saw a significant reduction in size of the tumor after 8 weeks. However, the tumors which were treated by RCF did not show any reduction in size. The difference was significant. The tumors treated by RCF were able to show necrosis, which is typical in tumours exposed to RCF. It is possible that the necrosis is caused by a lack of oxygen in the more invasive cancers.
In emf blocking , the findings suggest the fact that AM EMF exhibits anticancer properties in vitro as well as in vivo. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF triggers these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the growth of HCC tumors in the vivo.